Travis Kooima: 

  1. Identify the unique pathophysiologic mechanisms, clinical features, and diagnostic findings of autoimmune nodopathy/paranodopathy, especially in comparison to seronegative chronic inflammatory demyelinating polyneuropathy (CIDP)

  2. Explain the importance of the immunoglobulin G4 (IgG4) mediated inflammatory response seen in nodopathy/paranodopathy and its relationship to efficacy of specific immunomodulatory treatments.

Brandon Nidea:

  1. Demonstrate the historical progression of AI technology​

  2. Describe basic knowledge on how AI works​

  3. Illustrate the impact of AI in Epilepsy diagnostics​

  4. Outline the benefits of AI integration for patient care​

  5. Identify the limitations and barriers of deeper integration into current systems​

Session date: 
05/18/2026 - 4:00pm to 5:00pm CDT
Location: 
Virtually
United States
  • 1.00 AMA PRA Category 1 Credit
  • 1.00 University of Wisconsin–Madison Continuing Education Hours
    • 1.00 Approved for AMA PRA Category 1 Credit™
Please login or register to take this course.
Discloser List CME Internal Report